[Pokinimisi]
> [philips]
>In contrast, a 2007 analysis of medical journals from the period of the pandemic[12][13] found that the viral infection was no more aggressive than previous influenza strains. Instead, malnourishment, overcrowded medical camps and hospitals, and poor hygiene promoted bacterial superinfection. This superinfection killed most of the victims, typically after a somewhat prolonged death bed.[14][15]

Ni poznano, kaj je pobilo toliko ljudi, vse to so hipoteze. Ti pa delaš zaključke, ki po tvojem 100% držijo, kot da je to dejstvo. Pa ni. Alternativna razlaga je tudi tale, ki si jo kar sam citiral: malnourishment. Med vojno je bil zelo otežen dostop do kvalitetne hrane, ki vsebuje veliko hranil. V medicini in znanosti je pa zelo dobro poznano dejstvo, da ti največ dolgoročne škode naredi slaba prehrana v otroštvu in mladosti. Če si kolikor toliko kvalitetno hranjen v otroštvu, boš kot odrasel bolj zdrav, četudi ješ daljše obdobje slabšo hrano. tako da je čisto možno, da je tako visoko umrljivost med mlajšimi ljudmi povzročilo ravno to. Vendar, kot rečeno, ne vemo, zato je butasto, da hipoteze navajaš kot neizpodbitna dejstva.

Glej, dejstvo je da so nadpovprečno umirali mladi. In da zelo verjetno ni za smrtnosti kriv aspirin (s tem se je debata začela). Zelo podrobno je napisano na strani CDC:

wwwnc.cdc.gov/eid/ar...79_article

In izpostavljeno je tole:

Epidemiologic data on rates of clinical influenza by age, collected between 1900 and 1918, provide good evidence for the emergence of an antigenically novel influenza virus in 1918 (21). Jordan showed that from 1900 to 1917, the 5- to 15-year age group accounted for 11% of total influenza cases, while the >65-year age group accounted for 6% of influenza cases. But in 1918, cases in the 5- to 15-year-old group jumped to 25% of influenza cases (compatible with exposure to an antigenically novel virus strain), while the >65 age group only accounted for 0.6% of the influenza cases, findings consistent with previously acquired protective immunity caused by an identical or closely related viral protein to which older persons had once been exposed. Mortality data are in accord. In 1918, persons >75 years had lower influenza and pneumonia case-fatality rates than they had during the prepandemic period of 1911–1917. At the other end of the age spectrum (Figure 2), a high proportion of deaths in infancy and early childhood in 1918 mimics the age pattern, if not the mortality rate, of other influenza pandemics.